http://forum.eo.nl/mmbob/thread.jsp?for ... ge=8#reply
Nu is hier een neurologische studie dat Autisten een andere hippocampus hebbenMediamarty schreef: Ik heb deze site wel eens bestudeerd. Er blijft bij mij de vraag staan wat het betekend om "God in je hart" te hebben. Volgens mij betekend dat zoiets dat je je verstand moet uitschakelen ofzo. Ik kan het in iedergeval niet voelen. Mijn autisme zorgt er overigens ook voor dat ik geen relatie met mensen aan kan gaan, ik voel geen verbindingen. Aangezien god almachtig zou hij dit makkelijk kunnen compenseren met een betekenis volle droom of wat dan ook.
Ik vraag me dan ook sterk af. Als deze almachtige barmhartige god een arm schepsel ziet dat 25 jaar probeert met hem in contact te komen, kan hij dan niet een heel klein beetje meewerken? Het totaal afwezig blijven van antwoord kan volgens mij drie dingen betekenen:
A) Ik doe iets verkeerd. Dit zou inhouden dat alle hulp die ik gezocht heb mij niet kon helpen, oftewel, christendom moet een vreselijk complex geheel zijn. Lijk mij sterk, want er zijn mensen die hem plotseling "vinden". 25 jaar moet genoeg zijn om iets van een antwoord te vinden.
B) God vind mij niet aardig. Hij laat blijkbaar niks van zich horen hoe lang ik hem ook gezocht heb. Dit lijkt me sterk, want ik heb heel erg mijn best gedaan.
C) Geloof komt uit het onderbewuste en mensen die het gevoel missen kunnen niet geloven, oftwel god bestaat niet als autonome entiteit. Dit is wat mij betreft het meest logische antwoord of mis ik iets?
http://serendip.brynmawr.edu/bb/neuro/n ... ellew.html
Neurobiological Perspectives on Autism
Lauren Hellew
Autism is a pervasive developmental disorder characterized by restricted, repetitive, and stereotyped patterns of behavior, along with severe impairments in reciprocal social interaction, verbal and non-verbal communication, and cognitive development (1,2,3). If the brain is responsible for behavior then it should follow that disordered autistic behaviors should be explainable in terms of brain abnormalities and disordered neurobiological processes. While findings are generally speculative and the etiology of the disorder remains somewhat of an enigma, there is significant evidence that autism is associated with neurobiological dysfunction.
Autistic individuals are often highly socially withdrawn to the extent that they may appear to live in a world of their own. Infants typically fail to develop normal attachments to parents or caregivers. They may seem indifferent to other's gestures of affection towards them, and may even resist being held or otherwise engaged in physical or emotional interaction. They tend to make little or no use of eye contact, smiling, facial expressions, gestures, and other signals of social intent. They continue to manifest many of the same social impairments throughout childhood, adolescence, and adulthood. For example, they typically fail to develop normal peer relationships and they generally demonstrate an impairment in social-emotional reciprocity. Normal displays of emotion or empathy are uncommon: they do not generally offer comfort to others, nor do they seek others for comfort in their own times of distress. Similarly, they lack the ability to share in the enjoyment of other's pleasure and they resist sharing their own enjoyment with others.
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Evidence has also suggested that autism may be related to specific neurological damage to the limbic system (4,7). A great deal of research in this area has been conducted by Drs. Bauman and Kemper of Harvard Medical School and Boston University School of Medicine, respectively. These researchers have examined post-mortum brains of autistic individuals and have found the amygdala and the hippocampus to be underdeveloped. In particular, they have reported finding densely packed, unusually small neurons in the amygdala and hippocampus of autistic individuals. While the exact implications of these findings remain somewhat speculative, examination of the normal functions of these structures as well as related animal research may help to explain how such neurological damage may be connected to the traits and behaviors associated with autism.
The amygdala is thought to play a role in the control of aggression and emotion. This is significant in that autistics are often either overly aggressive or passive and may appear emotionless. When the amygdala is damaged or removed, animals exhibit social withdrawal, compulsive behaviors, failure to learn about dangerous situations, memory deficits, and difficulty adjusting to novel situations - characteristics that are similar to those seen in autism. The amygdala may also function in response to various types of sensory stimuli, another system which appears to function abnormally in autistics (7). The hippocampus is thought to be primarily responsible for learning and memory. It has been hypothesized by Dr. Bernard Rimland that autistic individuals have a specific cognitive deficit related to learning and memory. When the hippocampus is damaged or removed, animals demonstrate an inability to store new information into memory and they often display characteristics commonly seen in autism, including stereotypic, self-stimulatory behaviors, and hyperactivity (7).
hier nog iets over de relatie tussen hippocampus en TLE en godservaringen:
http://www.psychiatrictimes.com/p950927.html
Complex Partial Seizures Present Diagnostic Challenge
by Richard Restak, M.D.
September 1995, Vol. XII, Issue 9
Temporal lobe epilepsy (TLE), now more commonly called complex partial seizure disorder so as to include seizures that originate in the frontal foci, straddles the borderland between psychiatry and neurology. Since the condition may involve gross disorders of thought and emotion, patients with temporal lobe epilepsy frequently come to the attention of psychiatrists. But since symptoms may occur in the absence of generalized grand mal seizures, physicians may often fail to recognize the epileptic origin of the disorder. Indeed, misdiagnosis and failures of diagnosis are common in TLE. Fortunately, the illness is marked by certain "signature" symptoms that can aid in its identification.
John Hughlings Jackson observed in the late 1800s that seizures originating in the medial temporal lobe often result in a "dreamy state" involving vivid memory-like hallucinations sometimes accompanied by déjà vu or jamais vu (interpreting frequently encountered people, places or events as unfamiliar). Jackson wrote of "highly elaborated mental states, sometimes called intellectual aura," involving "dreams mixing up with present thoughts," a "double consciousness" and a "feeling of being somewhere else." While the "dreamy state" can occur in isolation, it is often accompanied by fear and a peculiar form of abdominal discomfort associated with loss of contact with surroundings, and automatisms involving the mouth and GI tract (licking, lip-smacking, grunting and other sounds).
Experiential Illusions
In the 1940s and '50s, Wilder Penfield, a neurosurgeon at the Montreal Neurological Institute, artificially elicited "dreamy states" by cortically stimulating the lateral temporal neocortex, the anterior hippocampus or the amygdala in awake epileptic patients prior to their surgical resections. During these operating room experiments, the patients experienced what Penfield referred to as "experiential illusions."
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